DISSERTATION APICAL PERIODONTITIS

Root canals of teeth with apical radiolucency associated with a draining sinus tract chronic apical abscess or suppurative apical periodontitis have been reported to harbor a mean number of 17 species By default, clicking on the export buttons will result in a download of the allowed maximum amount of items. However, there is no direct evidence showing that extraradicular bacteria were actually established as an extradicular infection independent of the intraradicular infection. Therefore, factors other than the mere presence of a given putative pathogenic species may play a role in the etiology of symptomatic endodontic infections 77 , Detection of Filifactor alocis in endodontic infections associated with different forms of periradicular diseases. Therefore, any species found in the canal has the potential to be an endodontic pathogen or at least to play a role in the ecology of the microbial community.

Microbial involvement with treatment failures is supported by two strong evidence-based arguments. Microbial examination of root canals and periapical tissues of human teeth. Herpesviruses in endodontic pathoses: In vivo evaluation of microbial reduction after chemo-mechanical preparation of human root canals containing necrotic pulp tissue. For instance, the acute apical abscess is usually dependent on the intraradicular infection, i. Strengthening of the association with apical periodontitis of some species previously recognized as candidate pathogens has also become evident by molecular findings.

As MR1 presents microbial riboflavin metabolite antigens, and CD1d presents endogenous or microbial glycolipid antigens, MAIT cells and iNKT cells survey the non-protein fraction of the potential antigenic universe.

dissertation apical periodontitis

The export option will allow you to export the current search results of the entered query to a file. Light and dissertxtion microscopic studies of root canal flora and periapical lesions. Microbial diversity in endodontic infections has been consistently assessed by anaerobic culturing and culture-independent molecular biology methods.

dissertation apical periodontitis

Endodontic infections develop in a previously sterile place which as such does not contain a normal microbiota. In another study, Siqueira et al.

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dissertation apical periodontitis

A high ;eriodontitis was observed for both interindividual samples from the same root region but from different patients and intraindividual samples from different regions of the same root comparisons. Identification and quantification of archaea involved in primary endodontic infections.

Phylogenetic analysis of bacterial and archaeal species in symptomatic and asymptomatic endodontic infections. The question as to whether the extraradicular infection is dependent on or independent of the intraradicular infections periodontitjs special relevance from a therapeutic standpoint.

The Association of Statin Intake and Healing of Apical Periodontitis After Root Canal Treatment

Genotypic microbial community profiling: Molecular analysis of persistent periradicular lesions and root ends reveals a diverse microbial profile. Bacteriology of dental abscesses of endodontic origin.

Apical periodontitis, an acute or chronic inflamination around the apex of the tooth, is caused by bacteria in the root canal. Chronic apical periodontitis Perioxontitis infections are conspicuously dominated by anaerobic bacteria organized in a mixed community.

Distinctive features of the microbiota associated with different forms of apical periodontitis

All these findings indicate that the microbiota of root canal-treated teeth with apical periodontitis is more complex than previously anticipated by culture studies.

Association of black-pigmented bacteria with endodontic infections. Histologic investigation of root canal-treated teeth with apical periodontitis: In other words, while associations of any specific species with any form of apical periodontitis is seldom, if ever, observed, the bacterial community profiles seem to follow some patterns related to the different presentations of apical periodontitis JavaScript is disabled for your browser.

In addition to frequency of detection, causation may be strengthened on the basis of potential pathogenicity in animal models or deduced from association with other human diseases.

Search for dissertations about: “apical periodontitis”

Identification of spirochetes treponemes in endodontic infections. Prevalence of yeasts in saliva and root canals of teeth associated with apical periodontitis. Periodontitid analysis of the human distal gut microbiome.

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First, there seems to be an increased risk of adverse treatment outcome when bacteria are present in the canal at the time of filling disserration — Therefore, any species found in the canal has the potential xpical be an endodontic pathogen or at least to play a role in the ecology of the microbial community.

However, while there is no reason based on solid scientific evidence to consider an infected canal as a focus of infection to distant body sites, except for periodohtitis compromised patients, the opposite should not be disregarded either, as there is no clear evidence to consider endodontic infections as segregate events with no effect on the rest of the body.

This review paper highlights the distinctive features of the endodontic microbiota associated with diverse clinical conditions. The extraradicular occurrence of several anaerobic bacteria has also been reported in post-treatment apical periodontitis lesions — Strengthening of the association with apical periodontitis of some species previously recognized as candidate pathogens has also become evident by molecular findings.

Distinctive features of the microbiota associated with different forms of apical periodontitis

Sixty teeth were examined, in 30 patients taking statins and 30 not taking statins control. Some canals of teeth with large lesions were found to harbor even more than 40 taxa Molecular and cultural analysis of the microflora associated with endodontic infections.

Bacteria in the apical root canal of teeth with primary apical periodontitis. The microbiota of acute apical abscesses. The mysterious appearance of enterococci in filled root canals.